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You continue with compressions and defibrillations and your partner places an advanced airway. The patient is a 54-year-old man who collapsed in front of his family after complaining of chest pain for several hours. On your arrival, first responders from the fire department are performing high-quality basic cardiac life support.
T-waves are quite tall and possibly peaked (HyperK?), but potassium returned normal. I do not see OMI here and all trops were only minimally elevated, consistent with either chronic injury from cardiomyopathy or with acute injury from sepsis. What is the QT interval? In LBBB, the QT interval is partly prolonged by the wide QRS. Bogossian et al. (1)
A patient had a cardiac arrest with ventricular fibrillation and was successfully defibrillated. IF the initial ECG following successful defibrillation shows evidence of acute OMI — such patients have much to gain from immediate cath with PCI. link] The COACT trial was fatally flawed (see below).
Josh Kimbrell, NRP @joshkimbre Judah Kreinbrook, EMT-P @JMedic2JDoc This is the first installment of a blog series showing how transcutaneous pacing (TCP) can be difficult, and how you can improve your skills. TCP In Transit: A case reviewing transcutaneous pacing, false electrical capture, and re-arrest.
15 minutes after EMS arrival, after at least 6 defibrillations, the patient achieved sustained ROSC. Meyers and Smith in the October 15, 2022 post of Dr. Smith's ECG Blog ). Written by Pendell Meyers A man in his 50s was found by his family in cardiac arrest of unknown duration. Restoration of sinus rhythm is evident in Figure-1.
He was defibrillated into VT. He then underwent dual sequential defibrillation into asystole. See these related cases: Cardiac arrest, defibrillated, diffuse ST depression and ST Elevation in aVR. This patient was witnessed by bystanders to collapse. They started CPR. EMS arrived and found him in Ventricular Fibrillation (VF).
Background Information: Double external defibrillation (DED) is an intervention often used to treat refractory ventricular fibrillation (RVF). This procedure involves applying another set of pads attached to a second defibrillator to a patient and shocking them in hopes of terminating the rhythm. N Engl J Med.
He required multiple defibrillations within a period of a few hours. This time, the arrhythmia did not spontaneously terminate — but rather degenerated to VFib, requiring defibrillation. Some episodes of PMVT would terminate spontaneously — but on many occasions, the PMVT degenerated to VFib, requiring defibrillation.
He was defibrillated, but they also noticed that he was being internally defibrillated and then found that he had an implantable ICD. He was unidentified and there were no records available After 7 shocks, he was successfully defibrillated and brought to the ED. There was no bystander CPR. Here is the initial ED ECG.
He underwent further standard resuscitation EXCEPT that we applied the Inspiratory Threshold Device ( ResQPod ) AND applied Dual Sequential Defibrillation (this simply means we applied 2 sets of pads, had 2 defib machines, and defibrillated with both with only a fraction of one second separating each defibrillation.
VF was refractory to amiodarone, lidocaine, double-sequential defibrillation, esmolol, etc. Then the patient would have been taken to the critical care area with a defibrillator at his side while waiting for the cath lab to be ready. He reports feeling nauseated with emesis. There is ischemic ST depression in V4-V6.
She was unable to be defibrillated but was cannulated and placed on ECMO in our Emergency Department (ECLS - extracorporeal life support). After good ECMO flow was established, she was successfully defibrillated. Here is a case of ECMO defibrillation with near shark fin that was due to proximal LAD occlusion. The K was normal.
Again, it is common to have an ECG that shows apparent subendocardial ischemia after resuscitation from cardiac arrest, after defibrillation, and after cardioversion. Much depends on the post resuscitation ECG and its evolution shortly after defibrillation. Does this patient have ACS? Should he necessarily go to the cath lab?
We also gave insulin and furosemide (which take much longer to have their effect) After episodes of asystole and VF with defibrillation, she obtained ROSC and this 12-lead was recorded: The monitor around this time was NOT showing a very wide complex What do you think?
Known cardiac defibrillator. appeared first on REBEL EM - Emergency Medicine Blog. Design: Prospective, observational, before and after implementation of a protocol study. Excluded: Obvious cause for OHCA prior to SDCT or on hospital arrival. Known obstructive coronary artery disease or known coronary stent. Pre-existing DNR order.
She was successfully revived after several rounds of ACLS including defibrillation and amiodarone. In our hope of increasing appreciation of the clinical insights that this fascinating phenomenon may provide — we highlight its occurrence in our blog posts. On arrival to the ED the patient was intubated with normal vital signs.
But because Dr. Mastoras recognized the hyperacute T waves, the patient was immediately seen, the polymorphic VT was immediately defibrillated, and the patient was rapidly diagnosed and treated. A healthy 45-year-old female presented with chest pain, with normal vitals. What do you think? There’s TWI in aVL but this is concordant to its QRS.
This episode self terminated before defibrillation was possible. For review of how Systematic 12-Lead ECG Interpretation not only increases accuracy, but also speeds you up — Please check out ECG Podcast #1 , wit h outline and link to my talk on this subject in the July 24, 2024 post in Dr. Smith's ECG Blog.
But artifact is "alive and well" — and learning to recognize it will amaze many of your colleagues ( and may serve to avoid an unnecessary defibrillation or two ). The red coloring is because the telemetry software incorrectly identified the artifact as VT. See if you can identify the underlying QRS complexes. Here is an example of that.
Adapted from Dr. Smith’s EKG Blog. Defibrillator pads are placed and you give 2 g IV magnesium over ten minutes. It is recommended to use an unsynchronized setting as the defibrillator may not be able to track the R-wave in the polymorphic waveform. An ECG is performed and is shown below: Figure 1. She denies chest pain.
Today's case reminds us of the intuitive logic that if a patient has a shockable arrest ( ie, VFib ) — and following successful defibrillation shows evidence of acute OMI ( even if STEMI criteria are not necessarily fulfilled ) — that such patients have much to gain from immediate cath with PCI. (
She spontaneously converted (Defibrillation was not performed). Most such rhythms in the setting of ischemia are VF and will not convert without defibrillation. The above ECGs show the initiation and continuation of a polymorphic ventricular tachycardia. After about 90 seconds of chest compressions she awoke.
Two hours later , the patient returned to this same urgent care facility with worsening chest pain, and this ECG was performed: While getting another ECG, the patient suffered cardiac arrest: After multiple defibrillations, ROSC was achieved with ongoing "STEMI". In this case, the EMS provider was routed to the urgent care facility.
The arrhythmia spontaneously converted before defibrillation was achieved. When you have identified the end of the QRS complex and the beginning of the ST segment you can assess whether there is concordant ST segment elevation, concordant ST depression or whether there are excessively discordant ST segments.
She was defibrillated successfully from ventricular fibrillation and developed a perfusing rhythm. A 56 yo f with h/o HTN and hypercholesterolemia called EMS from home after onset of L chest pain radiating to the left arm. Before EMS arrived, she had "seizure activity" and became unresponsive. She was intubated.
After amiodarone and several defibrillations and about 20 minutes after initial arrest, stable ROSC was achieved. ECG#1 There is a regular tachycardia with a ventricular rate of about 180 bpm. Smith comment : When there is a regular wide complex tachycardia, first assess whether it is sinus or not. At a rate of 180, this is unlikely.
She was given CRT-D (Cardiac Resynchronization Therapy-Defibrillator). For the majority of the tracing there are two P waves for each QRS. There is a LBBB. The PR interval gradually prolongs until a QRS is dropped and the pattern repeats itself. If there is no BBB the QRS will be narrow. There is for the most part 2:1 AV block.
Figure-2: A rapid method for estimating the QTc ( Figure from My Comment in the March 19, 2019 post in Dr. Smith's ECG Blog ). == Clinical Implications of a Short QTc: The differential diagnosis for today's tracing, with its short QTc ~360 msec. Treatment is by ICD ( implantable cardioverter defibrillator ). 1 for a rate ~75/min; 1.
After resuscitation and defibrillation , there were no more episodes of TdP. Below is the patient’s 12 lead ECG following defibrillation. There are no clear signs of OMI. The patient was admitted and put telemetry. While on telemetry monitoring he suffered cardiac arrest and was resuscitated. There is a prolonged QTc. There a two PVCs.
Background: There are only two interventions that have been proven in the medical literature to improved outcomes in cardiac arrest: high-quality CPR and early defibrillation. Over the years, we as a scientific community have worked extensively to find other interventions that improve outcomes. Impedance threshold device application.
ECG 2 Especially in the context of the first ECG, readers of this blog will readily appreciate the ST elevations and hyperacute T waves in II, III, aVF, V6, and to a lesser extent V5. They had difficulty describing their symptoms, but complained of severe weakness, nausea, vomiting, headache, and chest pain. His response was: "OMI.
EMS report was that the patient had unknown down time with unwitnessed arrest, found initially in VFib arrest, defibrillated x1 followed by PEA arrest alternating with asystolic arrest during transport. Upon closer inspection, the defibrillator's monitor had assigned a spike for every large T wave as well as every QRS complex.
As worrisome as the initial recording in leads I and II look — a glance at lead III should immediately reassure us that defibrillation is not needed! I thoroughly reviewed this concept ( as well as other helpful clues to the presence of artifact ) in My Comment — at the bottom of the page in the August 26, 2022 post in Dr. Smith’s ECG Blog.
She was found to be in ventricular fibrillation and was defibrillated 8 times without a single, even transient, conversion out of fibrillation. She was immediately intubated during continued compressions, then underwent a 9th defibrillation, which resulted in an organized rhythm at 42 minutes after initial arrest. It was stented.
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The patient had 2 ventricular fibrillation arrests during transport, but was immediately defibrillated both times, and was awake in the ED, when the following ECG was recorded: The ST elevation has mostly resolved on this ECG, and were it not for the arrest and the prehospital ECG, this would not be a slam dunk diagnosis.
He was resuscitated with chest compressions and defibrillation and 1 mg of epinephrine. This young male had ventricular fibrillation during a triathlon. On his bib it stated that he had a congenital heart disorder. He arrived in the emergency department hemodynamically stable. His initial ECG is shown here.
He was found in ventricular fibrillation and defibrillated, then brought to a local ED which does not have a cath lab. This blog is full of cases in which OMI that did not meet STEMI millimeter criteria were dismissed. Here is the initial ED ECG: This is pretty obviously and inferior posterior OMI, right?
But after reading this blog, you all know that most OMI do NOT meet STEMI criteria. Defibrillated out of v fib in the cath lab. I thank you for constantly updating your blog and allowing free open access education on EKG interpretation. This was the Marquette 12 SL GE algorithm The computer did not even mention the ST elevation.
She was never defibrillated. As was seen in this case — defibrillation and/or overdrive pacing may be needed. She was given 3 mg IV epinephrine and multiple rounds of ACLS over approximately 20 minutes. Her husband stated that she had not been feeling well in the past 2 weeks and c/o dizziness as well as diarrhea. What do you think?
The patient was put on Extracorporeal Life Support in the ED 3 hours after initial resuscitation, the core temp was 30° C and the patient was defibrillated with a single attempt. On arrival, CPR was continued and core temperature was measured at 18° C (64.4° A 12-lead ECG was recorded: There is sinus rhythm with RBBB and right axis deviation.
Schick is a PGY3 at the Brown Emergency Medicine Residency in Rhode Island. I remember Allie well from her days in the Research volunteer program at Hennepin. The article is edited by Smith. Title: Is it just hot in here or is it a OMI? See additional image at the bottom of this post. Could this be induced by elevated core temperature? F (rectal).
He was defibrillated immediately and had return of normal mental status. The most likely location for an occlusion producing OMI changes in the anterolateral and inferior leads is the proximal-to-mid LAD. This ECG can never be a baseline ECG. Cath lab activation was cancelled but the transfer was accepted for urgent cardiology evaluation.
We examined the effect of ibutilide, a class III antiarrhythmic agent, on the energy requirement for atrial defibrillation and assessed the value of this agent in facilitating cardioversion in patients with atrial fibrillation that is resistant to conventional transthoracic cardioversion. She was on no medications. Potassium was normal.
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