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Hyperthermia and ST Elevation

Dr. Smith's ECG Blog

A formal echocardiogram was completed the next day and again showed a normal ejection fraction without any focal wall motion abnormalities to suggest CAD. Our patient had a Brugada Type 1 pattern elicited by an elevated core temperature, which is also a documented phenomenon. There is no further workup at this time.

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STEMI with Life-Threatening Hypokalemia and Incessant Torsades de Pointes

Dr. Smith's ECG Blog

He had significant history of CAD with CABG x5, and repeat CABG x 2 as well as a subsequent PCI of the graft to the RCA (twice) and of the graft to the Diagonal. At cath, he immediately had incessant Torsades de Pointes requiring defibrillation 7 times and requiring placement of a transvenous pacer for overdrive pacing at a rate of 80.

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Management of STEMI (ST-Elevation Acute Myocardial Infarction)

ECG & Echo Learning

I B ECG monitoring should start immediately and a defibrillator must be ready. IIa C During hospital stay (after primary PCI) Either stress echo, CMR, SPECT, or PET may be used to assess myocardial ischaemia and viability, including in multivessel CAD. STEMI , ST-segment elevation acute myocardial infarction ).

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OMI? Subendocardial ischemia? Does it matter in this clinical context?

Dr. Smith's ECG Blog

The note documents that the first view of the LCX showed 99%, TIMI 2 flow, but then (before intervention) was seen to fully occlude in real time (100%, TIMI 0). Soon after the witnessed occlusion, the patient suffered ventricular fibrillation arrest, from which he was immediately resuscitated with 1 defibrillation. Pre-intervention.

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