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Resuscitated from ventricular fibrillation. Should the cath lab be activated?

Dr. Smith's ECG Blog

He was defibrillated into VT. He then underwent dual sequential defibrillation into asystole. But cardiac arrest is a period of near zero flow in the coronary arteries and causes SEVERE ischemia. See these related cases: Cardiac arrest, defibrillated, diffuse ST depression and ST Elevation in aVR. They started CPR.

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1 hour of CPR, then ECMO circulation, then successful defibrillation.

Dr. Smith's ECG Blog

She was unable to be defibrillated but was cannulated and placed on ECMO in our Emergency Department (ECLS - extracorporeal life support). After good ECMO flow was established, she was successfully defibrillated. Here is a case of ECMO defibrillation with near shark fin that was due to proximal LAD occlusion. The K was normal.

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A 50-something with chest pain.

Dr. Smith's ECG Blog

More past history: hypertension, tobacco use, coronary artery disease with two vessel PCI to the right coronary artery and circumflex artery several years prior. VF was refractory to amiodarone, lidocaine, double-sequential defibrillation, esmolol, etc. The 3rd inferior lead ( = lead II ) — shows definite ST-T wave flattening.

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What are treatment options for this rhythm, when all else fails?

Dr. Smith's ECG Blog

It shows a proximal LAD occlusion, in conjunction with a subtotally occluded LMCA ( Left Main Coronary Artery ). Upon contrast injection of the LMCA, the patient deteriorated, as the LMCA was severely diseased and flow to all coronary arteries ( LAD, LCx and RCA ) was compromised. There is no definite evidence of acute ischemia. (ie,

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A 20-something woman with cardiac arrest.

Dr. Smith's ECG Blog

When I saw the ECG of this patient I saw that there was definitely something "off". She spontaneously converted (Defibrillation was not performed). Most such rhythms in the setting of ischemia are VF and will not convert without defibrillation. A repeat magnesium level was not drawn prior to coronary angiography.

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What Lies Beneath

EMS 12-Lead

They are not premature, by definition. We can, therefore, put down the defibrillation pads, set aside the amiodarone, and look further at the ECG. The coronary angiogram revealed no critical stenosis, or acute plaque ulceration. The green arrows, however, do show premature complexes. potassium) were within normal parameter.

E-9-1-1 130
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Very fast regular tachycardia: 2 ECGs from the same patient. What is going on?

Dr. Smith's ECG Blog

There is definite change in the morphology of the waveforms and there is also significant change in the polarity of the QRS complexes in the precordial leads. After amiodarone and several defibrillations and about 20 minutes after initial arrest, stable ROSC was achieved. Other coronaries were normal. Is there OMI?