Dr. Smith's ECG Blog

JULY 27, 2023

On arrival to the ED, the patient was diaphoretic, tachycardic. I did not think it was due to ACS, but we ordered an ED ECG immediately: What do you think? I was not worried for a coronary etiology. Peak plasma concentration of amphetamines is rapid ( within minutes ) following inhalation or injection.

Coronary 52

Coronary ACS Naloxone Plasma 52

Dr. Smith's ECG Blog

APRIL 8, 2016

Here is his ED ECG: There is obvious infero-posterior STEMI. Internal potassium balance and the control of the plasma potassium concentration. Plasma potassium ([K] p ) was measured at 15-minute intervals during and after the infusion in 31 patients. Medics stated that he had not been taking his clopidogrel for 2 weeks.

STEMI 52

STEMI Plasma Defibrillator OR 52

EMDocs

FEBRUARY 3, 2025

SLE is considered an atypical risk factor for cardiovascular disease and acute coronary syndrome(8,9,11). Treatment requiresaggressive anticoagulation, glucocorticoids, plasma exchange, and intravenous immunoglobulin (IVIG)(27). This leads to adiffuse hypercoagulable state and widespread vascular dysfunction. Ann Rheum Dis.

ED 71

ED E-9-1-1 ALS OR 71

Dr. Smith's ECG Blog

FEBRUARY 6, 2025

During observation in the ED the patient had multiple self-terminating runs of Non-Sustained monomorphic Ventricular Tachycardia (NSVT). CT coronary angiogram showed a hypoplastic RCA and dominant LCx. The minimum effective plasma concentration of flecainide is about 200 ng/mL. No PVCs are seen. and Brugada syndrome.

Plasma 72

Plasma ACS OR Coronary 72

Don't Forget the Bubbles

MARCH 24, 2025

This is likely because Dexmed helps dampen the sympathetic response to perioperative stress, improving coronary artery perfusion. Later phase : As plasma levels fall, vasoconstriction decreases. How is Dexmedetomidine used in the ED? One of the most exciting areas for Dexmed in the ED is its availability in non-IV routes.

E-9-1-1 59

E-9-1-1 Emergency Department ALS Critical Care 59