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David Didlake Acute Care Nurse Practitioner Firefighter / Paramedic (Ret) @DidlakeDW Expert contribution by Dr Robert Herman @RobertHermanMD @PowerfulMedical (Chief Medical Officer) An adult male called 911 for new-onset epigastric burning. To which the lead paramedic replied, “Not cardiac; his symptoms are atypical. Is this OMI?
The paramedics achieve return of spontaneous circulation (ROSC) after CPR, advanced cardiac life support (ALCS), and Intubation. Acute coronary syndrome (ACS) is responsible for the majority (60%) of all OHCAs in patients. EMS arrives and finds the patient in monomorphic ventricular tachycardic (VT) cardiac arrest.
David Didlake Acute Care Nurse Practitioner Firefighter / Paramedic (ret) @DidlakeDW Expert commentary and peer review by Dr. Steve Smith [link] @smithECGBlog A 57 y/o Female with PMHx HTN, HLD, DM, and current use of tobacco products, presented to the ED with chest discomfort. It’s judicious, then, to arrange for coronary angiogram.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review and commentary by Dr. Steve Smith [link] @SmithECGblog It is early-summer, approximately 1330 hours, no cloud cover overhead, and 86 degrees with high humidity. Are these findings consistent with a particular coronary distribution?
Madden, Paramedic. It should be emphasized here that this is a presentation of high-pretest probability for Acute Coronary Syndrome (ACS). An interesting comment provided by Paramedic Madden is that a few team members initially interpreted the T wave presentation as hyperkalemia, as opposed to occlusive hyperacuity. Physiology.
Below is the first ECG recorded by paramedics after 2 hours of chest pain, interpreted by the machine as “possible inferior ischemia”. In isolation this ECG does not show OMI, but following the paramedic ECGs this indicates spontaneous LAD reperfusion. It’s unclear if the paramedic ECGs were seen or missed in the ED.
Paramedics provided another 3 sprays of nitro, and 6mg of morphine, which reduced but did not resolve the pain. Similarly, if a patient with known CAD presents with refractory ischemic chest pain, the ECG barely matters: the pre-test likelihood of acute coronary occlusion is so high that they need an emergent angiogram. Alencar et al.
She was known to have a history of poorly controlled COPD, AFib, and multivessel coronary disease. Pharmacology Review Digoxin is probably one of those medications vaguely recalled from paramedic school. David Didlake, FF/EMT-P, AG-ACNP @DidlakeDW An elder female presented to the ED with worsening shortness of breath.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review provided by Dr. Steve Smith [link] @SmithECGblog A 72 y/o Male experiences a syncopal episode while seated. Evaluation of T-wave morphology in patients with left bundle branch block and suspected acute coronary syndrome. 3] Meyers, H.
But they have an interest in ECGs, lots of exposure to OMI ECGs as well as normals and mimics, an interest in acute coronary occlusion, and a talent for seeing the subtle waveform findings of acute OMI. He was a paramedic at the time. Such proficient interpreters include health care assistants and EKG technicians.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review provided by Dr. Steve Smith @SmithECGblog I was conducting QA/QI on two very recent cases and was struck by the uniqueness of both. It’s important to stress the presence of a normal QRS (i.e., This ECG immediately struck me as an LAD occlusion.
David Didlake Acute Care Nurse Practitioner Firefighter / Paramedic @DidlakeDW A 50 y/o Male was taking his dog for a leisurely stroll through the park when he suddenly experienced new onset chest discomfort. A second 12 Lead ECG was recorded: This is a testament to the dynamic nature of coronary thrombosis and thrombolysis.
The coronary angiogram revealed no critical stenosis, or acute plaque ulceration. Takotsubo should be a diagnosis of exclusion after angiography reveals no obstructive coronary disease, and repeat Echo displays left ventricular recovery. Furthermore, pertinent electrolyte values (e.g. potassium) were within normal parameter.
But the paramedic and the ED physician in this case did not subscribe to this idea. It is far too premature to say that paramedics and physicians should not be bothered to interpret ECGs labelled as "normal" or "otherwise normal" by the computer algorithm. Thus, this is obvious STEMI(+) OMI until proven otherwise.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review by Dr. Stephen Smith @smithECGblog I was reviewing ECG’s in our LifeNet database and happened upon this one without any knowledge of clinical circumstances. Cardiology admitted him for observation with plans for next-day coronary angiogram.
First trop was 7,000ng/L (normal 25% of ‘Non-STEMI’ patients with delayed angiography have the exact same pathology of acute coronary occlusion. The new ACC expert consensus explains that: “STEMI ECG criteria on a standard 12-lead ECG alone will miss a significant minority of patients who have acute coronary occlusion. Take home 1.
Patient 1 : a 75 year old called paramedics with one day of left shoulder pain which migrated to the central chest, which was worse with deep breaths. The paramedic notes called STEMI into question: “EMS disagree with monitor for STEMI callout. Coronaries were normal, as was serial troponin. Vitals were normal.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Peer review provided by Dr. Steve Smith [link] @SmithECGBlog An adult female called 911 for chest discomfort and difficulty breathing. Cardiology was consulted, who advised to surveil a metabolic process as this did not strike them as acute coronary syndrome.
She went to angio and had normal coronaries. Paramedics found her semi-conscious, pale, cool, diaphoretic, tachypneic, very hypotensive. No d-dimer or CT pulmonary angiogram was done when they discovered that she had normal coronary arteries. I discussed the case with Cardiology will admit to their service."
This was shown to me by a very astute Hennepin paramedic. Although this comes from a Hennepin paramedic, the patient was not brought to Hennepin County Medical Center. It is important for cardiologists to realize that a paramedic may see something they do not. of this post. This is my reponse. This is not tribalism.
David Didlake Firefighter / Paramedic Acute Care Nurse Practitioner @DidlakeDW Expert commentary provided by Dr. Ken Grauer CASE 1 An 82 y/o Male called 911 for sudden onset dizziness while at rest. Upon arrival he was found alert and oriented, and without gross distress. He denied difficulty breathing, epigastric pain, or chest discomfort.
link] Case continued The conventional algorithm diagnosed STEMI and so did the paramedics. A Coronary angiogram from 8 years prior revealed that he had had an inferior posterior STEMI at the time due to 100% occlusion of the proximal RCA. It is not yet available, but this is your way to get on the list.
Cortland Ashbrook from Spokane County, Washington, sent this message: Hey doctor Smith, I wondered if you’d give me your opinion on these ECG tracings I took as a paramedic in the field? Electrocardiographic diagnosis of acute coronary Occlusion Myocardial Infarction in ventricular paced rhythm using the modified Sgarbossa criteria.
This case was provided by Spencer Schwartz, an outstanding paramedic at Hennepin EMS who is on Hennepin EMS's specialized "P3" team, a team that receives extra training in advanced procedures such as RSI, thoracostomy, vasopressors, and prehospital ultrasound. One need not have obstructive coronary disease to have occlusive thrombus!
Written by Jesse McLaren Two 70 year olds had acute chest pain with nausea and shortness of breath, and called paramedics. But these ECGs were from the same patient: #1 on paramedic arrival and #2 thirty minutes later. Thankfully this patient’s second ECG met STEMI criteria, so paramedics brought them as a code STEMI.
The ECG is determined to be non-diagnostic by the treating paramedic. The treating paramedic withholds aspirin. The paramedic, feeling a bit sheepish, asked me to review the case. There is no jugular venous distension or pitting edema. The cardiac monitor is attached. A 12-lead ECG is obtained. The patient refuses an IV.
Clinical Course The paramedic activated a “Code STEMI” alert and transported the patient nearly 50 miles to the closest tertiary medical center. The diagnostic coronary angiogram identified only minimal coronary artery disease, but there was a severely calcified, ‘immobile’ aortic valve. Look at the aortic outflow tract.
This case was sent by Lou B, a paramedic and RN. The coronaries were clean (this is not the gold standard, however, as some patients with ischemic ST elevation may have clean coronaries). ACTUAL CORONARY ANATOMY: Dominance: Right LM: A 5 mm vessel which bifurcates into the LAD and LCx coronary artery.
The paramedic recorded a series of ECGs; the initial ECG is representative here: Computer read: “ Normal ECG ” What do you think? The paramedic interpreted this as a STEMI. The Subtle STEMI calculation is used to differentiate a subtle anterior acute coronary occlusion from early repolarization (ER). or LAD occlusion?
The symptoms improved somewhat after the paramedic gave her nitroglycerin. In combination with the subtle ST depression in aVL , these changes are diagnostic for, or at least nearly so, for acute occlusion of a coronary artery, probably with some reperfusion, as inferior T-waves are inverted and the T-wave in aVL is reciprocally upright.
My most talented blog readers are paramedics because they have to put themselves on the line every time they activate the cath lab. In any case, these further support the diagnosis of coronary occlusion or near occlusion. The minute this medical student saw the first ECG, he knew the diagnosis without any further information.
Her initial 12-lead ECG that was obtained by paramedics in the field is shown in Figure-1. Figure-1: The initial ECG in todays case, obtained by paramedics in the field. ( NOTE: Because LBBB changes the sequence of LV repolarization it may be more difficult to recognize acute coronary occlusion on ECG. See text ). ( See text ).
The paramedics found the patient with ROSC and a GCS 7, and an ECG showing LBBB with possible lateral ST elevation. This ALONE is very strong evidence of acute coronary occlusion. The patient was brought to the ED as a possible Code STEMI and was seen directly by cardiology. Any indications for cath lab activation?
Pretest probability: Especially when there is no Chest pain, or there are very atypical symptoms, one should be very suspicious of the diagnosis of coronary occlusion unless the ECG is crystal clear. Outcome "I later found out that this is a patient who regularly calls paramedics to c/o chest pains and he had fooled many of them.
Pretest probability: Especially when there is no Chest pain, or there are very atypical symptoms, one should be very suspicious of the diagnosis of coronary occlusion unless the ECG is crystal clear. Outcome "I later found out that this is a patient who regularly calls paramedics to c/o chest pains and he had fooled many of them.
Oxygen Administration Oxygen is crucial for patients suffering from a heart attack as it can help increase the amount of oxygen available to the heart muscle, which may be deprived due to blocked coronary arteries. Nitroglycerin can help to dilate coronary arteries and improve blood flow to the heart muscle.
When the paramedics arrived, they obtained a 12 lead ECG and confirmed the unstable vital signs. Smith pointed out that while atropine may may result in slightly more oxygen demand, the increase in cardiac output and in blood pressure would increase overall coronary perfusion and decrease ischemia. Why is the patient in shock?
Here’s the paramedic ECG (digitized by PMcardio). According to the STEMI paradigm, the patient doesn’t have an acute coronary occlusion and doesn't need emergent reperfusion, so the paramedics can bring them to the ED for assessment, without involving cardiologists. HR 40, BP 135/70, RR16, O2 100%. What do you think?
Here is the written paramedic report available after all the events were over: Patient was seen by witnesses to become unresponsive. Moreover, when someone has immediate resuscitation of an arrest witnessed by paramedics, they rarely have a GCS of 3 (deep coma). Moreover, it does not follow a coronary distribution very well.
This was texted to me by a paramedic while I was out running one day: "54 yo male chest pain started at 1pm. The patient is a 54yo man with diabetes and a known history of coronary diseae ( prior stent placement ) — who presented with new CP ( C hest P ain ) — therefore very high likelihood for having an acute coronary event.
When an intensive care paramedic attended first, an additional 13 minutes was saved. Recommendations for ongoing implementation include upgrading ECG transmission capability and extending thrombolysis to all levels of paramedics. minutes shorter than pre-PHT. There were no significant adverse events.
She was found by paramedics with an oxygen saturation of 64%, but could not tolerate BiPAP during transport due to claustrophobia. The scan showed a bicuspid aortic valve with severe stenosis and coronary artery disease. Acute coronary occlusion and acute pulmonary edema can coexist. As her pain worsened, so did her dyspnea.
You’re me, and you’re in paramedic school, in the thick of the cardiology section. The ultimate goal is to optimize coronary perfusion pressure (CPP)—in other words, the amount of blood flow into the coronary arteries. When the heart is full, it puts pressure on the myocardium, compressing the coronary microvasculature.
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