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Background Information: Double external defibrillation (DED) is an intervention often used to treat refractory ventricular fibrillation (RVF). This procedure involves applying another set of pads attached to a second defibrillator to a patient and shocking them in hopes of terminating the rhythm. N Engl J Med.
2 Standard management for VT and VF involves the use of electrical defibrillation, high-quality chest compressions, and epinephrine. Initial guidelines defined “refractory” as VT or VF occurring despite three shocks from a cardiac defibrillator. Tips for use of dual sequence defibrillation 11 : Use the same model of defibrillator.
He was defibrillated into VT. He then underwent dual sequential defibrillation into asystole. See these related cases: Cardiac arrest, defibrillated, diffuse ST depression and ST Elevation in aVR. This patient was witnessed by bystanders to collapse. They started CPR. EMS arrived and found him in Ventricular Fibrillation (VF).
VF was refractory to amiodarone, lidocaine, double-sequential defibrillation, esmolol, etc. Suppose the OMI had been recognized, or suppose another ECG had been recorded and it showed definite OMI. But as it was, the delay to defibrillation was not long and it may be that nothing could have saved him.
She was unable to be defibrillated but was cannulated and placed on ECMO in our Emergency Department (ECLS - extracorporeal life support). After good ECMO flow was established, she was successfully defibrillated. Here is a case of ECMO defibrillation with near shark fin that was due to proximal LAD occlusion. The K was normal.
They had a difficult time getting a definitive airway pre-hospital. Key to survival is high-quality CPR and early defibrillation. Case: EMS arrive to your emergency department with a 68-year-old man post cardiac arrest patient. It required multiple attempts which caused several prolonged interruptions in CPR. What should you tell him?
He required multiple defibrillations within a period of a few hours. There is no definite evidence of acute ischemia. (ie, This time, the arrhythmia did not spontaneously terminate — but rather degenerated to VFib, requiring defibrillation. An ICD ( Implantable Cardioverter Defibrilator ) was placed prior to discharge.
When I saw the ECG of this patient I saw that there was definitely something "off". She spontaneously converted (Defibrillation was not performed). Most such rhythms in the setting of ischemia are VF and will not convert without defibrillation. Are these ECG changes related to the CNS infection perhaps?
Today's case reminds us of the intuitive logic that if a patient has a shockable arrest ( ie, VFib ) — and following successful defibrillation shows evidence of acute OMI ( even if STEMI criteria are not necessarily fulfilled ) — that such patients have much to gain from immediate cath with PCI. ( The April 8, 2022 post by Drs.
There is definite change in the morphology of the waveforms and there is also significant change in the polarity of the QRS complexes in the precordial leads. After amiodarone and several defibrillations and about 20 minutes after initial arrest, stable ROSC was achieved. In ECG #1 there was monophasic R-waves V2-V6.
They are not premature, by definition. We can, therefore, put down the defibrillation pads, set aside the amiodarone, and look further at the ECG. The blue arrows show RBBB morphology, and it’s tempting to deploy a compulsory diagnosis of PVC’s, but remember: these wide QRS complexes arrive subsequent to a regularly timed P wave.
She was found to be in ventricular fibrillation and was defibrillated 8 times without a single, even transient, conversion out of fibrillation. She was immediately intubated during continued compressions, then underwent a 9th defibrillation, which resulted in an organized rhythm at 42 minutes after initial arrest. It was stented.
She was successfully revived after several rounds of ACLS including defibrillation and amiodarone. E lectrical A lternans : Definition/Features/Mechanisms Electrical alternans — is a beat-to-beat variation in any one or more parts of the ECG recording. On arrival to the ED the patient was intubated with normal vital signs.
He was found in ventricular fibrillation and defibrillated, then brought to a local ED which does not have a cath lab. I would also measure the ST segments and determine if it was a STEMI by millimeter criteria (as outlined in the 4th Universal Definition of MI ), or OMI without STEMI. And the article was rejected.
Treatment is by ICD ( implantable cardioverter defibrillator ). Regarding Today's ECG — I chose the lead with the most ST-T wave deviation ( = lead V2 in Figure-1 ) to draw in 2 vertical BLUE lines highlighting the point where I thought definite “peaking of the T wave" is seen.
So this is a normal amount of STE in V2 and V3, defined by Universal Definition of MI as up to 2.0 So there is definitely no STEMI, and the STE is normal. Defibrillated out of v fib in the cath lab. The remarkable ECG findings are multiple: There are small-but-definite Q waves in each of the inferior leads ( RED arrows ).
She was never defibrillated. The ultimate reason for the long QT was never definitively determined. Drug-induced QT interval cannot be completely ruled out, but the tox consult found the she had definitely not overdosed and did not believe that therapeutic doses would do this. What do you think?
In the hope of reinforcing Dr. McLaren's case for immediate cath in today's patient — I thought it worthwhile to take another look at the subtle-but-definitely-present ECG findings that were clearly evident in the initial tracing ( Figure-1 ). First troponin I was 150 ng/L (normal <26 in males and <16 in females) and peak was 7,500.
She was defibrillated and resuscitated. To Emphasize: The reason definitive diagnosis is important in today's case — is that the senior ED physician interpreted ECG #1 as "nothing too exciting". Upon arrival to the emergency department, a senior emergency physician looked at the ECG and said "Nothing too exciting."
She was never seen to be in ventricular fibrillation and was never defibrillated. Medics found her apneic and pulseless, began CPR, and she was found to be in asystole. With ventilations and epinephrine, she regained a pulse. She was hypotensive in the ED and her bedside echo showed a normal RV and LV. BP gradually rose.
Cardioversion/defibrillation. This definition was changed following an expert consensus panel in 2013 — so that at the present time, all that is needed to diagnose Brugada Syndrome is a spontaneous or induced Brugada-1 ECG pattern, without need for additional criteria. Acute febrile illness. Variations in autonomic tone. Hypothermia.
As she has had an out of hospital arrest, the likely path would be (following cardiac work up) to be offered an implantable defibrillator (ICD). Depending on what you read and the exact definition applied to ‘recurrence’ of SVT somewhere between 30-50% of infants will not require therapy beyond 12-18 months.
We examined the effect of ibutilide, a class III antiarrhythmic agent, on the energy requirement for atrial defibrillation and assessed the value of this agent in facilitating cardioversion in patients with atrial fibrillation that is resistant to conventional transthoracic cardioversion. 25, 2022 ).
He was defibrillated immediately and had return of normal mental status. Cath lab activation was cancelled but the transfer was accepted for urgent cardiology evaluation. 40 minutes after the first ECG, just before transport, a repeat ECG was obtained: Continued, active OMI of the anterior, lateral, and inferior walls.
Rhythm C: This telemetry strip from an older adult was initially thought to need defibrillation. Rhythm B: This patient was seen in the ED — and thought to be in AFlutter with 4:1 AV conduction. Figure 1 – Examples of artifactual distortion ( excerpted from my ECG Blog ) — that resulted in arrhythmia misdiagnosis.
This definition was changed following an expert consensus panel in 2013 — so that at the present time, all that is needed to diagnose Brugada Syndrome is a spontaneous or induced Brugada-1 ECG pattern, without need for additional criteria.
You must also pass your practical examinations, which can include things like HARE traction splints, manual airway management, or proper CPR and AED (Automated External Defibrillator) use. In order to stay up to date, you must take something called a CEU, or Continuing Education Unit.
Treatment is by ICD ( implantable cardioverter defibrillator ). Regarding ECG #1 — I chose the lead with the most ST-T wave deviation ( = lead V2 in Figure-1 ) to draw in 2 vertical BLUE lines highlighting the point where I thought definite “peaking of the T wave" is seen.
Soon after the witnessed occlusion, the patient suffered ventricular fibrillation arrest, from which he was immediately resuscitated with 1 defibrillation. Final Diagnosis: "STEMI" (of course, as you can see in the ECGs above, this is not true, by definition this was NSTEMI. In other words, millimeters really don't matter!
The submitter started the patient on amiodarone and arranged implantation of a defibrillator. == MY Comment , by K EN G RAUER, MD ( 12/27 /2024 ): == Superb discussion by Dr. Frick in today's case, that highlights a series of important points regarding the ECG recognition of stable VT ( V entricular T achycardia ).
In this case, you should get a second defibrillator and perform double sequential external defibrillation (DSED). Simply attach a second defibrillator as shown in the diagram below and deliver max shocks from both devices simultaneously. In the second case, the patient never converted meaning the shock did not do its job at all.
Definitive care for an intrapartum cardiac arrest is a maternal perimortem cesarian section, which may be performed by any emergency physician. Sure, we still do CPR, defibrillate as needed, and give Epinephrine based upon our local guidance. These cases are incredibly sad and rarely yield survivable outcomes, for the mother or fetus.
Fortunately , you don't need to make a definite diagnosis. Second , when you have a rhythm problem, you are likely to be able to fix the problem with electricity (cardioversion, defibrillation, pacing). But the superior axis with positive QRS in V1 is difficult to reconcile with an accessory pathway.
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