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The patient was brought to the ED and had this ECG recorded: What do you think? Cardiac arrest #3: ST depression, Is it STEMI? After 1 mg of epinephrine they achieved ROSC. Total prehospital meds were epinephrine 1 mg x 3, amiodarone 300 mg and 100 mL of 8.4% sodium bicarbonate. And what do you want to do?
The cath lab was activated for STEMI. The patient had very poor IV access and bloods were only obtained just before leaving the ED for the cath lab. Hyperkalemia alone can cause inferior-posterior pseudo-STEMI: Notice that in both cases, the ST elevation is downsloping and the T-wave is inverted. There is still 1st degree AVB.
This post will focus on the key parts of the guideline that affect ED evaluation and management. Calcium is associated with harm but is still necessary in certain situations (hyperkalemia, calcium channel blocker overdose) (Level 3 recommendation: no benefit). Editorial Comment : Yes to PCI after arrest with STEMI on ECG.
Discharge Diagnosis was STEMI (The STE did not meet "criteria," so "OMI" would be better, but "STEMI" is far better than what this could have been called: NonSTEMI) Quotes from a note written by a really fine and knowledgable physician: "12-lead EKG was obtained initial 1 at time zero. "In
A New Seizure in a Healthy 20-something More cases of long QT not measured correctly by computer (these are all fascinating ECGs/cases): Bupropion Overdose Followed by Cardiac Arrest and, Later, ST Elevation. Is it STEMI? Chest pain in high risk patient. Are these Hyperacute T-waves? What is going on here? As per Drs.
Aligning Community Data for the Common Good by Michael Baker , MA, EFO, Fire Chief at Tulsa Fire Department and Justin Lemery , Director of EMS, Tulsa Fire Department discussed the far-reaching positive effects that reviewing data and creating community partnerships can have in overcoming homelessness, overdoses, and chronic illness.
This is pathognomonic of hyperkalemia (I suppose it could be due to a massive overdose of a sodium channel blocking drug, maybe). They transported to the ED. The history, obtained subsequently, is interesting: The patient had been seen at an outside ED 2 days prior and the K was 2.5 She was in shock with thready pulses.
Myocarditis is virtually indistinguishable in the ED from MI. See this case: Pericarditis, or Anterior STEMI? But absence of ST depression in aVL is not meant to rule out anterior MI! Positive troponin: this transformed the differential Dx from MI vs. pericarditis to MI vs. myocarditis. Confirmation bias? But confirming what?
Final Diagnosis: "STEMI" (of course, as you can see in the ECGs above, this is not true, by definition this was NSTEMI. But the "final diagnosis" commonly just reflects whether the patient was given emergent therapy or not, regardless of the definition of STEMI/NSTEMI). In other words, millimeters really don't matter!
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