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STEMI with Life-Threatening Hypokalemia and Incessant Torsades de Pointes

Dr. Smith's ECG Blog

Here is his ED ECG: There is obvious infero-posterior STEMI. What are you worried about in addition to his STEMI? Comments: STEMI with hypokalemia, especially with a long QT, puts the patient at very high risk of Torsades or Ventricular fibrillation (see many references, with abstracts, below). There is atrial fibrillation.

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Tasty Morsels of Critical Care 010 | Cardiac disease in pregnancy

Emergency Medicine Ireland

Pregnancy itself with its bump in plasma volume, reduced Hb and increased cardiac output is like one long exercise stress test. While interesting and exam worthy you’re still going to see STEMI on the ECH and have to go to the cath lab so perhaps it doesn’t change much of what we do.

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Patient with severe DKA, look at the ECG

Dr. Smith's ECG Blog

Discussion See this post: STEMI with Life-Threatening Hypokalemia and Incessant Torsades de Pointes I could find very little literature on the treatment of severe life-threatening hypokalemia. Internal potassium balance and the control of the plasma potassium concentration. and/or in the presence of acute MI. mEq/L to 3.0 mEq/L to 2.0

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Severe shock, obtunded, and a diagnostic prehospital ECG. Also: How did this happen?

Dr. Smith's ECG Blog

Internal potassium balance and the control of the plasma potassium concentration. to greatly decrease risk (although in STEMI, the optimal level is about 4.0-4.5 I could find very little literature on the treatment of severe life-threatening hypokalemia. The estimated deficit associated with a serum decrease from 4.0

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A comatose patient with a carbon monoxide level over 50%

Dr. Smith's ECG Blog

In fact, there is laboratory evidence that CO toxicity increases Plateletneutrophil aggregates and plasma myeloperoxidase (MPO) concentration and thus may precipitate ACS (though this is by no means clinically proven). Intravascular Neutrophil Activation Due to Carbon Monoxide Poisoning What do you think of this ECG?

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